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Galectin-3, Cardiac Function, and Fibrosis

  • University Medical Centre Groningen
  • University of Groningen
  • Sanataria de Navarra Research Institute

Research output: Contribution to journalEditorialAcademicpeer-review

19 Citations (Scopus)

Abstract

This Correspondence relates to the article by Frunza et al (Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac Fibrosis. Am J Pathol 2016, 186:1114-1127).

Original languageEnglish
Pages (from-to)2232-2234
Number of pages3
JournalAmerican Journal of Pathology
Volume186
Issue number8
DOIs
Publication statusPublished - 1 Aug 2016
Externally publishedYes

Bibliographical note

Publisher Copyright:
© 2016 American Society for Investigative Pathology

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