GM-CSF and specific type 2 cytokines induce CD103+and CD301b+cell states in cDC1s and cDC2s

  • Lukas Amon*
  • , Damir Vurnek
  • , Anna Seichter
  • , Giorgi Tchitashvili
  • , Tomasz Kaszubowski
  • , Markus Mroz
  • , Nincy Debeuf
  • , Tina Vogler
  • , Nicole Küpper
  • , Kaushikk Ravi Rengarajan
  • , Lukas Lächele
  • , Nounagnon R. Tochoedo
  • , Anna Baranska
  • , Stella E. Autenrieth
  • , Falk Nimmerjahn
  • , Kai Hildner
  • , Klaus Pfeffer
  • , Barbara U. Schraml
  • , Lukas Heger
  • , Bart N. Lambrecht
  • Christian H.K. Lehmann, Diana Dudziak*
*Corresponding author for this work

Research output: Contribution to journalArticleAcademicpeer-review

Abstract

The heterogeneity of conventional dendritic cells type 1 (cDC1s) and type 2 (cDC2s) is well established, yet the identity and origin of CD301b+cDC2s remain debated. Here, we show that CD301b+cDC2s and CD103+cDC1s develop from pre-committed progenitors in response to granulocyte/macrophage colony-stimulating factor (GM-CSF). While CD103+cDC1s acquire their phenotype and functional properties through GM-CSF-driven differentiation from pre-cDC1s, CD301b+cDC2s emerge as cytokine-induced states from DC2- and DC3-committed progenitors. CD103+cDC1s and CD301b+cDC2s exhibit enhanced T cell priming capacities and distinct cytokine expression profiles upon GM-CSF exposure. In vivo, DC-intrinsic GM-CSF sensing is dispensable for acquiring CD103 and CD301b expression with the notable exception of lung DCs, while specific type 2 cytokines induce CD103 and CD301b ex vivo. These findings identify GM-CSF and specific type 2 cytokines as central regulators of cDC1 and cDC2 effector differentiation and establish CD301b as a marker of a cytokine-driven cDC2 state.

Original languageEnglish
Article number116589
JournalCell Reports
Volume44
Issue number12
DOIs
Publication statusPublished - 23 Dec 2025

Bibliographical note

Publisher Copyright:
© 2025 The Authors. Published by Elsevier Inc. This is an open access article under the CC BY license. http://creativecommons.org/licenses/by/4.0/

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