Potassium channels in control of renal function

Pedro H. Imenez Silva; Carsten A. Wagner

doi:10.1016/j.kint.2019.10.029

Potassium channels in control of renal function

Pedro H. Imenez Silva, Carsten A. Wagner^*

^*Corresponding author for this work

Research output: Contribution to journal › Comment/Letter to the editor › Popular

3 Citations (Scopus)

Abstract

Potassium channels are important to control membrane potential and drive epithelial transport processes. In this issue of Kidney International, Bignon et al. report the role of the Kir4.2 K⁺-channel, localized at the basolateral membrane of proximal tubules, in the reabsorption of bicarbonate and the modulation of renal ammoniagenesis. The findings have implications for our understanding of how the kidney reacts to hypokalemia, an acid load, and the metabolic acidosis of patients with advanced stages of chronic kidney disease.

Original language	English
Pages (from-to)	253-255
Number of pages	3
Journal	Kidney International
Volume	97
Issue number	2
DOIs	https://doi.org/10.1016/j.kint.2019.10.029
Publication status	Published - Feb 2020
Externally published	Yes

Bibliographical note

Publisher Copyright:
© 2020 International Society of Nephrology

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10.1016/j.kint.2019.10.029

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abstract = "Potassium channels are important to control membrane potential and drive epithelial transport processes. In this issue of Kidney International, Bignon et al. report the role of the Kir4.2 K+-channel, localized at the basolateral membrane of proximal tubules, in the reabsorption of bicarbonate and the modulation of renal ammoniagenesis. The findings have implications for our understanding of how the kidney reacts to hypokalemia, an acid load, and the metabolic acidosis of patients with advanced stages of chronic kidney disease.",

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AU - Wagner, Carsten A.

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AB - Potassium channels are important to control membrane potential and drive epithelial transport processes. In this issue of Kidney International, Bignon et al. report the role of the Kir4.2 K+-channel, localized at the basolateral membrane of proximal tubules, in the reabsorption of bicarbonate and the modulation of renal ammoniagenesis. The findings have implications for our understanding of how the kidney reacts to hypokalemia, an acid load, and the metabolic acidosis of patients with advanced stages of chronic kidney disease.

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DO - 10.1016/j.kint.2019.10.029

M3 - Comment/Letter to the editor

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VL - 97

SP - 253

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Potassium channels in control of renal function

Abstract

Bibliographical note

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